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PERSPECTIVES ON THE PANDEMIC 
EPISODE #10
JUDY MIKOVITS & ROBERT KENNEDY JR. (Part 1 of 3)  

Perspectives on the Pandemic interviewed Dr. Judy Mikovits on April 16th, 2020, and again on May 15th. In her second interview, she was joined by Robert F. Kennedy, Jr. We have divided the two conversations into three parts, the first two focusing on Dr. Mikovits, and the third on Mr. Kennedy. This is part one. 

Auto-generated text with timestamps:

00:37
Judy there's been an almost
00:38
unprecedented onslaught against do
00:40
multiple articles in the mainstream
00:42
press from the Washington Post to Forbes
00:44
have come out to attack you in the
00:46
segments of the film planned emic that
00:48
you appeared in I want to separate your
00:49
story from the film itself as the two
00:51
have been conflated I want to start just
00:54
by asking mr. Kennedy you recently wrote
00:56
the introduction to Judy's book Plague
00:58
of corruption and in that you play Stern
01:01
a long line of dissident scientists from
01:03
Rachel Carson who discovered the dangers
01:06
of DDT to Alice Stewart who found that
01:08
x-rays during pregnancy led to
01:10
carcinomas and children later on
01:11
researchers who were first scorned and
01:14
later hailed as heroes
01:15
what makes Judy a dissident in this
01:17
tradition and what is the Semmelweis
01:19
effect
01:19
let's analyze effect as a is that
01:23
dynamic a predominant medical
01:29
orthodoxies are idiot and
01:38
reaction a medical community and
01:42
particularly doctors and the and the
01:46
medical cartel in the medical community
01:49
and go immediately into a defense
01:53
posture to defend things like you know
01:58
x-raying pregnant women even long after
02:02
the establishment between that procedure
02:05
in cancers and those children is well
02:07
established the same thing happened with
02:09
solidified the same thing happened with
02:12
he's counting all which was mercury
02:15
based his power powder that was for a
02:17
hundred years
02:18
only terminated at 1950 illnesses and
02:23
children DDT was another thing that the
02:27
American Medical Association because it
02:30
had recommended DDT for the suppression
02:32
of malaria it was last a the last group
02:37
to really to be willing to back off and
02:39
say yeah there's a problem here even
02:42
when the science was well well establish
02:45
ignite Semmelweis was a Hungarian and
02:50
a doctor at a time and most well more
02:55
than half of the women who went into
02:57
hospitals we're dying for a childbirth
03:00
we're dying of peripheral fever which
03:03
was also known as birth bed fever and he
03:07
had an idea this was before Pasteur our
03:10
before germ theory before anybody knew
03:11
about germs but he had an idea from his
03:15
own anecdotal observations that the
03:18
scientists who were the doctors were the
03:21
same doctors who were performing
03:22
autopsies that they were not cleaning
03:26
their hands between the autopsies and
03:28
delivering babies and that they were
03:30
bringing some kind of toxic particle
03:34
into the birthing bones and he did an
03:38
experiment and where he got them to wash
03:40
their hands
03:41
any drop birth and fever from around 50%
03:44
mortality from around 50 percent to
03:46
around 2 percent of women who came to
03:48
the hospital when he published on that
03:50
at first he was applauded and suddenly
03:54
he was derided and ultimately he was
03:57
tricked into going into a mental
03:59
institution and apparently murdered and
04:02
the medical associations at that time
04:05
what against him with a vengeance
04:08
because a challenge there are kind of
04:12
deified position as physicians I saying
04:17
something that you are doing instead of
04:20
helping patients it's actually hurting
04:22
them and that's part of the reason that
04:25
we see this you know this enormous
04:27
reaction to any kind of criticism of
04:30
vaccines and of course you know Judy's
04:34
science was a was a devastating blow and
04:40
because what you need to discover among
04:43
many other things what judy discovered
04:46
was that there was a virus that had been
04:50
grown on mouse brain tissue and you when
04:53
you grow a vaccine when you're making
04:55
you grow them on animal tissues on on
04:58
cocker spaniels on mice on pangolins
05:02
on bats
05:03
on horses and on insects and unhuman
05:07
lung tissue from aborted fetuses
05:10
that's how vaccines are made most
05:12
vaccines or many vaccines and Judy
05:16
discovered a virus where she was
05:19
assigned to work for the look for the
05:22
cause of chronic fatigue syndrome
05:24
chronic fatigue syndrome was a
05:27
devastating illness that appeared in an
05:30
epidemic form in the 1980s and it
05:32
affected mainly women and a medical
05:35
community behaved reprehensibly a
05:38
dismiss the disease as a psychosomatic
05:41
illness it was at a time when women were
05:44
first entering the job force high
05:46
positions and corporations and the this
05:50
illness is because not biologically
05:54
equipped to handle that kind of
05:56
responsibility that was traditionally
05:58
belonged to men was a province of men
06:00
and that that's why they're getting
06:03
these psychosomatic breakdowns and what
06:06
Judy found when she actually started
06:08
studying the disease for NIH is that the
06:12
women who complain of chronic fatigue
06:14
senator men had been diagnosed 63% of
06:19
them had a virus that she was able to
06:22
find identify in an isolate it was call
06:24
the XMRV virus and that only 4% of the
06:30
control group so of healthy women had
06:33
that had that incident that virus in
06:36
their blood she published on that and
06:40
NIH and Tony pouchy who was her boss was
06:43
okay with her publishing about it
06:45
because it didn't threaten any of their
06:46
paradigms subsequently Judy learned um
06:52
work that she did and from work that
06:54
other scientists were doing it by the
06:56
way XMRV by then had already been
06:59
associated with many other diseases
07:00
leukemias and cancer there's a lot of
07:03
really bad illnesses very very strong
07:06
associations and what Judy found out was
07:11
at the source of the xmrv not in the
07:15
blood supply
07:17
was coming originally a vaccine that had
07:21
been made a polio vaccine that had been
07:24
made using mouse brain tissue and it had
07:27
it was a virus that was common in mice
07:31
and it had made the leap to human beings
07:34
through vaccination and now it was not
07:36
only in that polio vaccine it was and
07:38
hepatitis A vaccine it was in the MMR
07:41
vaccine and it had contaminated the
07:45
entire blood supply
07:46
and that was something that Tony found
07:49
she couldn't broach because it was a it
07:53
was essentially an attack on his
07:56
industry it was saying that vaccines and
08:00
NIH had blessed and anointed and part of
08:04
the approval process at FDA had urged
08:08
CDC to recommend and mandate for 74
08:12
million children annually they had made
08:15
a very very bad mistake and instead of
08:18
saving lives those vaccines were most
08:22
likely to be causing more mortality than
08:27
they were averting and that is a threat
08:30
to the entire vaccine paradigm and their
08:33
only response to that threat was that
08:35
Judy make it as had to be destroyed
08:37
I'm Judy a Mike Ovitz I graduated from
08:41
the University of Virginia with a degree
08:43
in chemistry in 1980 went directly to
08:47
the National Cancer Institute in
08:49
Frederick Maryland where I was part of
08:51
the team that that made the first immune
08:55
therapy that was interferon alpha and it
08:58
was a curative therapy for hairy cell
09:01
leukemia from there I will join the
09:04
biological response modifiers program in
09:07
1983 where I worked under the direction
09:10
of dr. Frank resetti who discovered the
09:14
first human cancer-causing retrovirus
09:18
and and in cytokines and immune therapy
09:22
there we continued to work with AIDS
09:26
patients I was part of the team that
09:28
isolated HIV from saliva and
09:31
that confirmed Nobel laureate Luc
09:35
montagnier 'he's discovery and of HIV as
09:39
a possible causative agent of AIDS from
09:46
1987 to 91 I worked on my PhD thesis
09:52
which was award-winning in 1991 it
09:58
changed the paradigm for HIV AIDS as it
10:01
said the t-cell the cell that was killed
10:03
in hiv/aids was was not the target of
10:08
therapy it was actually the monocyte
10:09
macrophage which was the orchestrator of
10:12
the immune response so the idea that you
10:16
get damaged from a distance when a virus
10:18
infects and that you can have HIV and
10:21
not get AIDS if you protect the
10:25
activation of a dormant virus keep it
10:28
from being activated after in my
10:31
postdoctoral studies I worked in another
10:34
laboratory at the National Cancer
10:35
Institute learning molecular virology
10:37
under dr. Dave der C and what we did
10:41
there was along with Johns Hopkins dr.
10:45
Stephen Balin we looked at other
10:47
mechanisms by which viruses dis regulate
10:51
them make abnormal the immune response
10:54
because it's not the virus the infection
10:58
that is deadly it's the it's the immune
11:01
response it's the overactive immune
11:03
response or in an in a susceptible
11:06
individual that leads to the damage of
11:08
the tissue and ultimately in some cases
11:11
death in 1999 I accepted the job as
11:16
director of the lab of antiviral drug
11:19
mechanisms at Fort Detrick there again
11:22
in the National Cancer Institute that
11:25
was internationally recognized
11:29
laboratory for excellence and my job
11:32
there was to build a team to look at
11:34
AIDS associated malignancies AIDS
11:38
associated cancer we made seminal
11:41
discoveries in that in treatment in
11:44
developing treatment
11:45
for aids at that time I then moved to
11:49
California in 2001 where I directed the
11:54
cancer biology program of epigenetics
11:57
pharmaceuticals in Santa Barbara
11:59
California and that was taking those
12:02
discoveries and those medicines and
12:04
everything we'd learned in the previous
12:06
25 years in applying them to the
12:09
development of drugs and diagnostics for
12:13
a aged cancer associated with the
12:17
mechanisms we've discovered what is you
12:20
Sam red when were you there and what was
12:23
your role during the time that we were
12:25
looking at retroviruses and their in in
12:29
my postdoctoral studies and their effect
12:30
on the immune system I worked with a
12:35
collaborative effort with you Samara
12:38
that's the US Army Research Institute
12:41
for infectious diseases and it's right
12:44
there at Fort Detrick literally right
12:46
across the baseball field from my
12:49
laboratory in the National Cancer
12:51
Institute so what my job was in that
12:54
collaboration was to look at the Ebola
12:58
virus these ie restrain the highly
13:00
pathogenic Ebola virus that was deadly
13:04
and my job was to teach the Ebola virus
13:08
to infect human cells without killing
13:12
them and so I or animal cells to find a
13:16
cell line to grow the virus so that we
13:20
could study it because you can't study a
13:22
virus unless it's an obligate parasite
13:25
you need to have a host it must grow in
13:27
cells so you can grow up a lot of the
13:29
virus and and and study it and study how
13:33
it causes disease that I did that I
13:37
found several cell lines that the Ebola
13:41
virus would would live in and grow quite
13:43
happily without killing them and from
13:46
there my job was to try to understand
13:48
the difference between the very
13:50
pathogenic Zaire strain and a harmless
13:54
strain of Ebola virus called Ebola virus
13:59
restin so what I did was infected
14:01
primary white blood cells fresh from a
14:05
patient an individual person an
14:07
uninfected healthy person and I'd take
14:10
those and I didn't fetch with one one
14:13
with the pathogenic strain and one with
14:15
the non-pathogenic strain and I'd
14:17
subtract the difference what was the
14:19
disease signature we were looking at
14:22
what was that immune response well what
14:24
was the flame the fire that destroyed
14:26
the tissue and caused the disease the
14:29
microvasculature the bleeding the blood
14:32
disorders what what were those signals
14:35
and so from that we developed a
14:38
signature of disease and that's critical
14:41
because I went on to do signatures of
14:44
disease for certain types of
14:46
non-hodgkin's lymphomas we would call
14:49
these categories just non-hodgkins
14:52
lymphoma and the treatment really
14:53
matters if you understand what the
14:57
dysfunction is why the cancer developed
15:00
why the lymphoma developed and and that
15:02
not necessarily having anything to do
15:05
with infection it's just a signature of
15:07
infection or disease so back to teaching
15:12
the Ebola strain to infect healthy cells
15:16
what what is gain-of-function as it
15:20
relates to virology what is that what
15:23
you were doing at that time or or was it
15:26
attenuation and what what is the
15:28
difference and can you just describe
15:29
those two concepts so what I was doing
15:34
at the time that I was teaching Ebola to
15:38
infect human cells is largely considered
15:40
to be gain-of-function studies because
15:44
normally Ebola is another virus that we
15:48
get from bats and and it doesn't infect
15:52
human cells so I'm gaining a function
15:55
because I'm trying to teach it to infect
15:57
human cells and and and so that it can
16:00
live happily in human cells so yeah
16:04
if I were doing studies to attenuate the
16:07
virus that is make it weaker which we
16:11
would do
16:12
in order to make vaccines is I was I
16:15
would continuously pass it through
16:18
another animal not necessarily a human
16:22
but another animal because the immune
16:25
system when it sees a virus and an
16:27
infection it gives you that inflammatory
16:30
response and it tries to suppress just
16:34
to stop the expression of the virus a
16:36
silent virus is not a problem to your
16:39
immune system or the host so you don't
16:42
want it to divide and replicate and
16:45
build reservoirs no doctor quickly to
16:48
gain-of-function studies can you tell us
16:50
what did Francis Collins do I believe in
16:53
2013 and then what got reversed can you
16:56
just talk about that right so a number
16:59
of I religious I'm in Wayne Hobson and
17:01
others our colleagues said you know it's
17:03
really very dangerous to do this type of
17:06
work and so they advised Francis Collins
17:10
the head of the NIH you know to
17:12
literally put a moratorium on doing any
17:15
you know a law that said it was illegal
17:19
in this country to do gain-of-function
17:21
studies to do that kind of culturing
17:24
that that I did back in 99 because it
17:27
became clear to us that the Ebola
17:31
outbreak that killed 21,000 Liberians in
17:35
2014 was almost certainly the zyre
17:39
strain with many mutations that came by
17:44
way of Fort Detrick in in the US and I
17:48
think that was they were intentionally
17:49
spread or you think that was accidental
17:51
or I think it was I think it was
17:53
accidental but that was covered up then
17:55
and this was 2014 but certainly there
17:59
were more than 300 mutations in that
18:02
Zaire strain that weren't there in you
18:06
know prior to the manipulation in the
18:08
laboratory at that time so so sorry go
18:12
ahead and tell me yes so what so there
18:14
was a recommendation you were saying so
18:16
that so so that was at the time when
18:19
when all of that happened there was a
18:20
recommendation by Francis Collins and he
18:23
made it into a federal law I believe
18:26
that said you can't do that type or fund
18:29
that type of research in the United
18:31
States because it's too dangerous the
18:33
possibility that something an agent
18:36
could be released that's far more
18:38
dangerous and cause a worldwide pandemic
18:40
could occur so when Tony foutch he
18:43
funded these studies throughout that
18:45
illegal time period that's a problem in
18:50
many ways so you're saying he continued
18:52
to do gain-of-function studies that it
18:56
was illegal he continued to fund them so
18:59
he he funded the the Wuhan researchers
19:02
in collaboration with the North Carolina
19:05
researchers I don't believe the North
19:08
Carolina researchers had a biosafety
19:10
level 4 so that work was largely done in
19:14
Wuhan but Tony Fauci funded it and then
19:18
in 2017 mysteriously the ban was lifted
19:22
and and most people didn't know about it
19:24
at the time and only learned about it
19:26
looking at this latest outbreak how
19:28
could this happen we had a law against
19:30
this so I guess now is a good time if
19:32
you could tell us what is the process by
19:38
which vaccines are manufacturers and
19:42
what in particular is
19:43
xenotransplantation is if you could just
19:47
describe that process okay so the
19:52
process at least in in the last few
19:55
decades by which vaccines are
19:59
manufactured is to take those cell lines
20:04
that you have a virus growing in whether
20:07
the tissue the animal tissue that you
20:10
grow the virus in again it can't you
20:12
can't make a lot of virus for the
20:14
purpose of making the antigen in in a
20:17
vaccine without growing up large
20:20
quantities of it and for that you need
20:22
those animal or human cell lines or in
20:26
fact Mouse cell lines we we use Pig cell
20:29
lines it really depends you know for
20:32
most of my career what I did was make
20:35
cell lines how my job is to cells don't
20:39
grow out of the
20:40
side of the body very long because that
20:43
that's a definition of cancer so we we
20:45
transform cells from primary tissue and
20:49
make it into cell lines so there's
20:53
that's where we grow viruses so
20:55
xenotransplantation is the term that we
21:00
use for any time that you put foreign
21:03
tissue in another animal or anytime you
21:07
mix foreign tissue so technically if you
21:12
do injecting animal tissue into humans
21:17
into human blood in a vaccine is
21:20
xenotransplantation also we would do
21:24
surgery where we may take a pig a or
21:27
tick valve and replace somebody with
21:30
with heart surgery with a pig valve and
21:32
in the case of HIV patients we didn't do
21:37
that research because we recognize that
21:40
a dormant virus in the pig could become
21:43
pathogenic to somebody infected with
21:47
another kind of human retroviruses or
21:49
other viruses of the same family and
21:52
cause a tremendous disease well since
21:57
you brought them up please describe what
22:00
is a retrovirus and what makes it
22:02
different from a regular virus well a
22:07
retrovirus is an RNA virus we have a
22:12
large many families of RNA viruses
22:15
influenza viruses or RNA viruses corona
22:18
viruses or RNA viruses many many RNA
22:21
viruses that means they're nucleic acid
22:24
their their genomic their blueprint is
22:27
RNA not DNA like human blueprint so an
22:31
RNA virus or a virus writes its RNA
22:37
blueprint backwards reverse transcribe
22:41
so they call it retro so you write your
22:43
RNA backwards where you make DNA and
22:46
then you take then that it has another
22:50
enzyme called integrase that literally
22:52
cuts
22:53
open the DNA in the cell that the
22:56
retrovirus infects and inserts itself so
22:59
every time that cell is replicated say
23:03
blood cells every time you need to
23:05
respond to an infection or respond to
23:08
stimulations
23:09
every day we turn over 10 to the ninth
23:13
ten billion blood cells so those get
23:18
nucleated blood cells get integrated and
23:21
you're making a factory of virus so the
23:25
corona virus or influenza viruses aren't
23:28
retroviruses in that they don't
23:30
integrate into your cells into the cells
23:33
of the host so when the cell dies the
23:36
virus can't persist essentially all
23:38
animals have retroviruses in their
23:41
genome eight percent of the the human
23:45
genome is built up of retroviruses all
23:50
animals have retroviruses but they're
23:53
crippled so that they're not expressed
23:55
so they don't cause disease in the host
23:57
what's the difference between
23:59
xenotransplantation and zone OSIS zone
24:04
OSIS is that process of an animal virus
24:10
Xue infecting another animal through a
24:15
natural evolution let's just say that
24:19
the meat isn't properly cooked
24:22
you know pork and what the meat isn't
24:25
properly cooked so you don't kill the
24:27
pathogen and you eat that and and you it
24:30
can infect you or or you cut yourself in
24:34
the case of you know some of the
24:36
theories of how HIV jumped into humans
24:39
from animals so zoonosis is just the
24:42
process of evolution of a virus from its
24:47
original species to now be able to
24:50
infect another type of animal who is
24:54
Frank Crosetti and what did he initially
24:58
discover and then what did you discover
25:00
with him so dr. Frank Crosetti
25:04
discovered the
25:06
the first family of disease-causing
25:10
retroviruses and it's called human
25:14
t-cell leukemia lymphoma virus that
25:18
family so this wasn't known in 1980 when
25:22
Bernie poised and frank resetti isolated
25:25
the viruses from people with a very
25:26
aggressive cancer called adult t-cell
25:29
leukemia Frank also discovered
25:33
interleukin 2 5:15 and tgf-beta a key
25:38
cytokine that's a master regulator of
25:41
the hematopoietic the blood stem cell so
25:45
that's that's really your entire
25:47
adaptive immune system is what Frank
25:51
Crosetti discovered and without under
25:53
without discovering these interleukins
25:55
these factors that are communication
25:57
interleukin means communicate between
26:00
white blood cells and without
26:02
understanding the the conversation
26:04
between different subsets of cells you
26:08
you can't understand why a cancer occurs
26:11
an ogre overgrowth of those T cells so
26:14
very aggressive very fast-growing cancer
26:18
a deadly cancer and it's causative
26:21
because every human who has htlv has
26:27
adult t-cell leukemia not necessarily in
26:30
the cancer cell but in their body so
26:33
somehow indirectly largely htlv
26:37
contributes to cancer and that's why
26:40
it's causative tell me what you guys
26:43
discovered and what you published in the
26:46
journal science in 2009 yeah well in in
26:52
2006 I moved from epidemics
26:55
Pharmaceuticals to build to literally
26:59
design the first neuro-immune disease
27:03
institute and that using a systems
27:06
biology approach the same one we've used
27:09
for our entire careers we were studying
27:11
the disease chronic fatigue syndrome
27:14
because people with chronic fatigue
27:16
syndrome have a lot of what we call
27:19
opportunistic in fact
27:20
things that healthy people don't get
27:22
they have a lot of immune inflammatory
27:27
sick well I they have cytokine storms
27:30
just wanna pause really quickly can you
27:32
just tell us what are cytokines and what
27:35
is a cytokine storm a Saito Saito means
27:40
cells and cytokines is just the term
27:43
that that immunologist gave to the
27:47
communicating molecules the mediators
27:50
between cells in the immune system and a
27:53
cytokine storm is a large number of
27:56
cytokines let's just say we're a number
27:59
of cytokines five to seven cytokines
28:01
that are expressed together at the same
28:05
time and they're your army to go out and
28:09
fight an invader so that when that that
28:12
disease signature that that we've been
28:15
discussing is is a cytokine storm that
28:19
when when it when a virus is pathogenic
28:22
infecting a human you get the storm if
28:25
the site if the disease if the virus is
28:27
not pathogenic there's no there's no
28:30
expression of the cytokines because your
28:32
immune system says that's harmless we
28:35
don't need to worry about that but when
28:37
when there's something the immune system
28:39
needs to target it the communicators
28:42
between the cells are cytokines so one
28:45
of the first things I did when I met the
28:47
patients as I did entire family studies
28:50
and you see a lot of cancers in the
28:52
family you see a lot of autism you see a
28:55
lot of what we call neuro immune
28:57
diseases Parkinson's Alzheimer's things
28:59
like that as you follow the family and
29:02
you look at the environment for what
29:04
toxin could have caused the disease and
29:06
then I did these kinds of signatures so
29:09
the first thing that we did when we
29:11
looked for when we form the Institute is
29:13
go to whole families of some of the
29:16
sickest people with chronic fatigue
29:18
syndrome and and we found they had a
29:22
disease signature suggestive of a
29:25
retrovirus and interestingly enough that
29:28
retrovirus had been described in in men
29:33
with
29:33
a very aggressive prostate cancers so
29:37
you remember when I was at Johns Hopkins
29:39
University my job at the lab of
29:42
antiviral drug mechanisms was look at
29:45
retroviral associated cancers in one of
29:49
those was the very aggressive the most
29:52
aggressive of prostate cancer so I knew
29:55
of that work in 2005 and so we we
29:59
essentially began the process of trying
30:02
of hiding the retroviruses from the
30:05
sickest of patients and then getting the
30:08
sequence and the diagnostics and trends
30:10
transmitting them or looking at family
30:12
members and seeing if the disease got
30:15
the same cytokine storm the same disease
30:18
signature that that work on that
30:21
transmission in the isolation of that
30:24
new family of viruses in association
30:27
with a highly associated I think there
30:30
were probably nine zeroes in front of
30:33
the one highly highly highly
30:35
statistically significant that XM RV's
30:39
we called them or that's what the group
30:41
that ice that discovered the sequences
30:44
in prostate cancer they called it zina
30:47
tropic Zeno meaning four and murine
30:50
leukemia virus related virus so there's
30:53
a mouse cancer-causing virus that's
30:56
associated now not only with prostate
30:59
cancer but when we published this paper
31:01
in science October 8 2009
31:04
now with connecting cancers to
31:08
neurological diseases which of course we
31:10
saw with hiv/aids some some patients
31:14
would get cancer some would get
31:16
neurologic disease where did you
31:18
discover the XMRV and and and what and
31:22
and tell me run me through what the
31:23
implications were well we isolated it
31:26
from patients the most severe ill with
31:30
chronic fatigue syndrome it the XMRV had
31:34
been described by derice ian Silverman
31:37
but they're not virologist and they
31:39
didn't isolate the virus so until you
31:42
isolate the virus and and you show it
31:44
can be transmitted to a
31:47
their white blood cells it's there
31:49
they're a series of things called Koch's
31:51
postulates or Hills criteria
32:01
you
32:07
so this was really a big deal a new
32:11
family of disease-causing retroviruses
32:14
that could take you know one new virus
32:18
how many old diseases was the editorial
32:22
that John coffin wrote that that
32:24
accompanied this really widely
32:26
celebrated article in in science because
32:29
how many new diseases do we now have
32:32
instant therapeutics for which everybody
32:36
mainly thought these people were you
32:38
know you're just chronic fatigue
32:39
syndrome you're just tired no this is
32:43
inflammation of the spinal cord and
32:45
brain and their brains don't work their
32:47
muscles don't work they get a lot of
32:49
infections serious diseases that now we
32:51
actually have a therapeutic target it's
32:54
huge
32:55
to do think about that as soon as that
32:59
paper came out and we were immediately
33:01
contacted by our colleagues at the
33:04
National Heart and Blood Institute heart
33:07
blood and Lung Institute at NIH and we
33:10
started day one that paper came out with
33:14
testing the blood supply's developing
33:16
tests to test the blood supply because
33:18
of course we knew from our experience
33:21
with HIV that a contaminated blood
33:23
supply was how HIV spread through
33:27
populations that we were unaware of
33:31
being susceptible to the viral infection
33:34
so the the blood supply as in those
33:38
studies in in 2011 a march 29th at the
33:43
New York Academy of Sciences I showed a
33:46
presentation to show that in fact the
33:49
blood supply was heavily contaminated up
33:51
to 10 percent and but as dr. resetti
33:55
always taught me so the minute we
33:57
started the work to identify that the
34:00
blood supply was heavily contaminated we
34:03
a company contacted us and said we have
34:07
a technology that would decontaminate it
34:09
and in fact it did so we did side by
34:12
side parallel studies all along to make
34:14
sure we had a solution when I presented
34:17
those data March 29th of 2011
34:22
but during this time in in the two years
34:25
after the paper was published a number
34:28
of other troubling disease associations
34:31
came up first many cancers chronic
34:34
lymphocytic leukemia multiple myeloma
34:37
and in these were not necessarily just
34:40
us but colleagues of ours everyone
34:43
started to study the virus and it became
34:45
clear this infection was much more
34:48
widespread than simply the three million
34:50
Americans that at that time had a
34:53
diagnosis of chronic fatigue syndrome so
34:56
you know so manaus like Alzheimer's
35:01
disease like Parkinson's disease Lou
35:03
Gehrig's disease which had long we've
35:05
long had evidence of retroviral
35:08
involvement in a number of inflammatory
35:10
what we call idiopathic diseases because
35:14
we don't understand the pathogenesis how
35:16
you know what what is associated what
35:19
causes the disease so we that work
35:22
became quite troubling because of the
35:26
cost you know that that a contaminated
35:29
blood supply is what spread this
35:33
infection through a population and then
35:36
even more troubling was one of our
35:39
colleagues published a paper in January
35:42
an opinion paper in a journal called
35:44
frontiers in microbiology in January of
35:49
2011 and and what that paper said is one
35:53
of the most widely distributed
35:57
biological products where Mouse tissues
36:01
are used are vaccines and so it is
36:04
possible that the virus stocks when
36:07
we're growing up let's just say polio
36:10
virus in cell lines or we used Mouse
36:14
tissues back in the 1930s to attenuate
36:17
the polio virus the Royal we and then
36:21
and then in the in recent decades we
36:23
used viral monkey kidney cells to make
36:26
the polio vaccine well you cannot remove
36:29
their other RNA viruses or you've
36:33
removed
36:33
your antigen so these particles as dr.
36:38
Berke out was his name are former AG
36:40
colleagues as he wrote Ben Burke out
36:42
he said one you know it is possible that
36:45
the way Mouse viruses entered the human
36:48
viral was the particles were present in
36:51
the vaccines and that's when that was
36:57
the beginning in the end of my career
36:59
who are as I knew it what would be the
37:04
implication to government agencies and
37:07
to the pharmaceutical industry if in
37:11
fact these murine and other retroviruses
37:15
were being spread through the
37:18
vaccination regimen
37:20
well the input the implication is that
37:25
the really the vaccine program should
37:28
have ended right then and when when when
37:32
we discovered this in 2011 everything
37:34
should have stopped total moratorium
37:37
until we can figure this out because our
37:41
economy couldn't afford the liability
37:44
that that a you know via a contaminated
37:49
blood supply or contaminated vaccines
37:51
because our work was never to look
37:52
directly at the vaccines we did the
37:55
blood supply work so the implications
37:57
that chronic diseases that are exploding
38:01
in our world as we know it came from
38:04
vaccines yes as doctor as Hillary
38:08
Johnson wrote in the foreword of our
38:10
first book plague a disease to affect
38:13
the economy of nations so the
38:16
implications are economically just huge
38:19
like it was you couldn't I don't think
38:22
the country could have survived that
38:25
implication especially given the fraud
38:28
in the denial so your work your work was
38:31
not about like oh this is now we now
38:34
know it's in the vaccine supply or this
38:35
is originated from vaccines correct my
38:38
work was about the blood supply and
38:39
liking in it to the AIDS epidemic and
38:42
and that was the big the big deal
38:44
because the AIDS epidemic really was the
38:47
star
38:47
the Celaeno season so in fact it's burke
38:50
outs work that makes the connection to
38:52
vaccines with XMRV and this well this
38:55
was an opinion paper it exactly so I
38:59
mean what I'm hearing is is that there
39:01
would have been this massive exposure
39:04
massive liability there would have been
39:06
a clear implication that the that that
39:10
the vaccine regimen as we knew it was
39:15
unsafe but you didn't start out what was
39:19
your what was your feeling towards
39:21
vaccination when you began this research
39:24
well from my whole life as I told you
39:28
that from the first day in 1980 for 40
39:31
years it was my the hypothesis we worked
39:35
with is that we could teach the immune
39:38
system we could educate the immune
39:42
system to prevent or treat cancer and
39:46
infectious disease if we understood how
39:49
they caused pathogenesis of how they
39:51
cause disease so that immune therapy is
39:55
is a vaccine and so I'm not at all
39:58
anti-vaccine I encouraged my siblings to
40:03
have their children get the Gardasil
40:06
vaccine because of course at that time I
40:09
had no idea I'm not in vaccine
40:13
manufacturer I didn't know about the
40:17
paper that that Bob Burke out for in in
40:21
2011 I had no idea I had no idea my work
40:25
in implicated vaccines I was worried
40:28
about the blood supply because that was
40:30
still 25 million Americans and and we
40:34
did one of the studies in the UK and it
40:36
was 4% in the control groups and that's
40:39
a lot of people that that the UK and in
40:42
the US have to compensate at the level
40:45
and give the kinds of benefits we gave
40:48
HIV infected individuals in in the 80s
40:51
and 90s and to this day so I'm I'm not
40:55
anti vaccine therapy but you know
40:59
interferon alpha
41:01
is your mitigation right now for any RNA
41:05
virus in fact we could make a safe
41:07
vaccine right now using interferon alpha
41:12
using the other drug that we developed
41:15
in what 1986 with Candace pert known as
41:21
peptide T well the FDA kept peptide T
41:25
coming from coming to the market and
41:27
what peptide T is is stopped that
41:30
receptor known as ccr5 from sticking
41:34
like velcro to the white blood cell
41:37
so for coronavirus right now we could
41:40
take interferon alpha and peptide T and
41:45
and in transiently block a receptor
41:49
called cannabinoid receptor 2 that that
41:52
doesn't dampen isn't the dimmer switch
41:54
on the immune system so if we can just
41:57
stop the flame from getting too high we
42:00
just put that in a capsule and I could
42:02
put purified virus no RNA no DNA whole
42:06
virus particles take it as a capsule
42:08
keep it away from the lungs presented
42:11
present the antigen in a natural form to
42:14
the immune system in the bone marrow and
42:16
you'd have forever and this is plug in
42:19
play every RNA virus we can present it
42:23
to the right resident stem cells where
42:26
we need it to respond where the disease
42:29
where the tissue injury that where we
42:31
want them to respond so if if our if our
42:35
work hadn't been censored and we didn't
42:37
just stop everything the day we realized
42:40
this as a matter of fact we realize this
42:43
at a July 22nd 2009 invitation-only
42:47
meeting at that the NIH held and and in
42:52
that meeting the the big oh my god was
42:56
that the lab workers were cero
42:59
converting meaning developing antibodies
43:02
and so because they were developing
43:05
antibodies it meant that all Mouse
43:09
research had to be biosafety level 3
43:13
like we worked with with a
43:15
Ivy that is the double gloves and you
43:17
protect yourself with the HEPA filtered
43:19
air flow and you autoclave you burn out
43:22
all the trash you know it's it's very
43:24
simple and in fact I used biosafety
43:28
level three measures when I was working
43:31
in the Nevada Institute be in our
43:34
Institute in Nevada because we didn't
43:36
have a biosafety level 3 facility and so
43:39
what the government did the the the
43:42
title of the chapter in our first book
43:44
plague I think it's chapter a is you
43:49
know the July 22nd invitation-only
43:52
meeting and after I finish my talk which
43:56
showed cancers and neuro immune disease
43:59
chronic fatigue syndrome and then some
44:01
childhood diseases one called
44:04
niemann-pick which is a child like
44:06
Alzheimer's so some of these diseases
44:09
that children shouldn't get today
44:11
associated with just that one virus
44:14
because we didn't realize it was a whole
44:16
family of viruses at that time the the
44:20
people at the meeting said oh my god you
44:22
know the heads of our Institute said oh
44:24
my god and I'm thinking oh thank you
44:26
they saw what I saw but it wasn't oh my
44:29
god it was oh my god we can't afford to
44:32
retrofit every Mouse facility in the
44:36
country
44:36
and make it biosafety level 3 we can't
44:40
afford to protect the lab workers and so
44:45
I got infected in 2010 many many many of
44:51
my colleagues who worked with mice when
44:54
we didn't realize that these viruses
44:56
could aerosolize so what I say in the
44:59
book is contagious cancer you know
45:02
literally I can cough on you cancer you
45:06
know and if you're susceptible enough
45:09
you develop it soon and if you're not
45:11
you develop it later or not at all or if
45:14
you use the measures that we know of
45:16
that we know protected AIDS patients
45:19
from getting disease of course you would
45:23
never get illness but what the
45:24
government did was cover it up and
45:28
refused
45:29
use the drugs to the patients so they
45:32
literally took curative therapies and
45:34
the FDA said oh that's not approve for
45:37
that you can't use that off-label and
45:41
and so many of you know friends and
45:43
colleagues are literally dead before
45:46
before 60 Judy isn't the the base
45:49
problem here what's going on in these
45:52
these labs these biosafety level 3 4
45:56
whatever labs they or or the ones that
45:58
aren't taking precautions but is it
46:01
isn't the whole issue this
46:03
xenotransplantation attenuation
46:06
gain-of-function cycling viruses through
46:10
you know the animal tissue I mean what
46:13
what what is the basis of that is that
46:17
the basis of our problem yeah
46:20
the basis of our problem is essentially
46:22
every medicine we make is using you know
46:27
Mouse tissues biological therapies
46:30
growing up in cell lines this is I would
46:33
say since the 90s my first job when I
46:36
was a natural products chemist
46:38
so I isolated chemo therapies from
46:41
plants and and I'm back to doing that
46:43
again from the cannabis plan and from
46:46
other natural plants from Chinese herbs
46:48
we're going back to natural medicine
46:50
this would just simply say we need to
46:53
stop every bit of the technologies all
46:56
the so right tuxie Mavs ma B means
47:00
monoclonal antibody you made it in a
47:02
mouse you made it in another human
47:04
tissue
47:04
we have aborted fetal tissue in these
47:07
vaccines well other human tissue in in
47:10
another human is developed gonna develop
47:13
autoimmunity so you can see so the
47:16
entire industry the pharmaceutical
47:18
industry would stop today or should have
47:21
stopped in 2011 can you just briefly
47:24
take us through the the different xeno
47:28
transfers that are going on just a quick
47:30
list including the aborted fetal cells
47:34
and if you could just go into a little
47:35
bit more depth about why the immune
47:37
system rejects
47:38
other human cell lines in these vaccines
47:41
but just you know I've heard you in
47:43
interviews just kind of bullet point how
47:45
you know the different animal tissues
47:47
that are used in different products
47:50
right so essentially every gene therapy
47:54
product the vectors that we've been
47:57
using and I'll just mention the cancer
47:59
drug everybody's it's called cart T
48:03
chimeric antigen receptor T cells and
48:06
I'm sure it was on Time magazine for a
48:09
few years
48:09
oh this curative therapy well that car T
48:12
cell therapy where we manipulate human
48:15
tissues and we take your own cancers out
48:18
and make a chimeric antigen receptor do
48:21
we change your own T cells and put it
48:23
back that's made on a murine leukemia
48:25
virus vector so that's how we get that
48:28
in which what's meereen mouse mouse so
48:34
that's made on a mouse retrovirus the
48:37
ones we discovered it's made on these
48:39
Mir Mouse virus vectors so gene
48:42
therapies would have to stop the way
48:45
we're doing them now because those are
48:47
biologics that are doing
48:49
xenotransplantation essentially every
48:52
single vaccine has at least one animal
48:59
tissue in it or another human tissue so
49:02
birds the flu vaccines are made in birds
49:06
so there's at least one retrovirus and
49:09
many other RNA viruses coronaviruses
49:12
everyone all the animals have corona
49:15
viruses just like all animals have
49:17
retroviruses so the flu vaccine that was
49:20
used in in Italy had for live influenza
49:25
live attenuated influenza viruses
49:28
including h1n1 all of which caused upper
49:32
respiratory infections and that one was
49:34
made in dog cells dog kidney cells so
49:38
you have brought in other viruses you
49:41
know potentially corona viruses here in
49:44
the United States the vaccines are made
49:46
in chicken you know we grow the the
49:49
antigens the the viruses in check
49:52
and is the idea that these viruses are
49:54
retroviruses in the host animal are are
49:59
fine for the host animal but something
50:01
happens you know they're potentially
50:03
carcinogenic or whatever they are in us
50:05
is that the idea yes so retroviruses we
50:08
have our own endogenous retroviruses and
50:10
they don't kill us and the same things
50:12
true so for instance simian immune
50:15
deficiency virus is the retrovirus that
50:18
jumped theoretically into humans causing
50:21
AIDS so that's the HIV work we did so
50:25
when you jump species a virus that's at
50:28
home in that's xeno that's in the animal
50:31
it's it's called a commensal just like
50:34
our microbiome we have so many micro
50:37
micro organisms that are we called it
50:39
good bacteria
50:40
it helps us it's at equilibrium with the
50:43
host it's no problem are the immune
50:46
system of the host has it but every time
50:48
you inspect something foreign into an
50:53
immune compromised the very young the
50:56
very old and of course people with
50:59
genetic primary immune deficiencies many
51:03
of which we don't even know because the
51:04
proteins and the immune system is so
51:06
large so it's an that's that's a simple
51:09
answer to xenotransplantation is
51:12
anything foreign put in your body from
51:16
another animal species and those do and
51:18
can cause disease and that's dr.
51:22
Rossetti's discoveries of 1983 in 1991
51:27
at the time of my PhD thesis HIV was 1
51:31
million Americans and the group act up
51:34
and others got drugs you sooner you
51:37
remember it was a similar story to what
51:39
we're seeing today oh don't use that
51:41
danger as a Z T or this or that drug you
51:45
know and and the disease continued just
51:48
like we're hearing today with Kovan 19
51:50
the the mitigation measures should have
51:53
been interferon alpha which costs you
51:56
know like 50 cents a docent for n yuca a
51:59
$600 vial could protect a thousand you
52:03
know of the elderly of the most
52:05
susceptible
52:06
so nobody never needed lockdown if you
52:09
use that an interferon alpha and then
52:12
hydroxychloroquine which we knew was
52:15
also a valuable therapy for viruses
52:18
coming from bats even in Ebola to stop
52:21
the 2014 outbreak that was used in the
52:25
doctor that got infected
52:27
what are corona viruses and then why do
52:29
you think this is a SARS Co v2 plus xmrv
52:34
what what makes you think that in terms
52:36
of the presentation of the illness
52:37
well corona viruses are RNA viruses and
52:41
they have as we know an envelope that
52:43
you protect the nucleic acid by building
52:46
a fatty acid protein envelope around it
52:48
it looks like that little particle forms
52:51
so the RNA is the blueprint of the virus
52:54
and as I mentioned retroviruses are a
52:57
different blueprint and RNA DNA
52:59
blueprint just subtly difference but
53:02
they're also envelope viruses so when
53:05
you look when you do a test for
53:08
polymerase chain reaction what you're
53:11
doing is you're taking a small piece
53:13
let's just say 10% of the blueprint 150
53:18
base pairs out of 8,000 and you're
53:20
amplifying it artificially in the lab
53:23
and and making millions of copies out of
53:27
that one copy and then you call it a
53:30
positive and so as you know the PCR
53:33
tests that are being done now you do a
53:36
nasal swab or a throat swab and scrape
53:38
the tissue this cells that the virus
53:41
would infect the epithelial cells out of
53:43
the nasal passages that's where corona
53:45
viruses live when you do that it doesn't
53:49
say an infectious virus at all it just
53:51
says a piece of RNA that in order to see
53:53
it you had you had to amplify a zillion
53:57
times in in that quick short time period
54:00
of the test so that the the serology
54:04
testing actually says you've been
54:07
infected because the virus got in your
54:09
blood you didn't have the the
54:11
degradation the machinery in your nose
54:14
and you were susceptible and you did
54:16
because of other infections you you
54:19
actually got
54:20
an infection that your immune system
54:22
couldn't handle and you made an antibody
54:24
and that's that's the principle of all
54:27
vaccines you give them the antigen in a
54:30
low dose and they make an immune
54:32
response to it an antibody and then the
54:34
next time they see the pathogen they
54:37
don't have to go through that two-day
54:39
process of make the antibodies and the
54:41
virus can't build enough of a reservoir
54:43
so you're immediately giving the
54:46
antibody the next time it sees the it
54:49
sees the pathogen you don't get as sick
54:51
or maybe you don't get sick at all so
54:53
that is a vaccine strategy and that's
54:56
why I said the people that have the
54:58
antibodies are already immune they don't
55:00
need to worry about a vaccine let me
55:03
yeah let me just ask you that they're
55:04
saying that they're not sure if the
55:07
people who have had this or have the
55:09
antibodies are going to be immune
55:11
doesn't that an undermine the whole
55:13
argument behind vaccination exactly and
55:17
and you know that is the own so the only
55:21
criteria of vaccination is do you
55:24
develop an antibody it's propaganda
55:27
masquerading as science this isn't
55:30
science no if you make an antibody I've
55:32
heard things like well the antibody
55:34
testing says and what an IgG means that
55:38
antibody family means a past infection
55:41
and that would help us to say that this
55:43
virus has been in this country a lot
55:45
longer than they say it's been in this
55:47
country and then the second thing is if
55:50
you make an IgM antibody it's a recent
55:54
or an acute infection and then you're
55:56
not necessarily immune so you need to be
55:59
protected in a way if you've been
56:02
exposed recently so now I'm hearing this
56:06
I'm seeing in the press and seeing
56:09
people say oh the IgG antibody is a
56:13
later stage of disease and it's not a
56:17
later stage of disease at all it's it's
56:19
immunity so we're playing this word game
56:22
and we're using serology positive which
56:25
should say we've developed a herd
56:27
immunity and we've developed a
56:29
population that can go back to work
56:31
because in fact there
56:33
going to protect the rest of us in the
56:35
most vulnerable in in the in the u.s.
56:38
but now that that science is being
56:40
twisted to say it says something it
56:42
doesn't say the media continues to
56:45
report that we have no evidence that
56:46
patients who survived coronavirus have
56:49
immunity I think actually the truth is
56:51
the opposite we have no evidence that
56:54
survivors of coronavirus don't have
56:55
immunity and a great deal of evidence to
56:57
suggest that they do the question of
57:00
immunity is linked to health policy and
57:01
that workers who have gained immunity
57:03
can be a strong part of our economic
57:05
recovery the silver lining to so many
57:08
infections in the meat processing
57:10
industry is that a large portion of
57:13
these workers now have immunity those
57:15
workers should be reassured that they
57:18
likely won't get it again instead of
57:19
being alarmed by media reports that
57:21
there is no evidence of immunity you've
57:24
stated publicly that you'd bet at all
57:25
that survivors of coronavirus have some
57:27
form of immunity
57:28
can you help set the record straight
57:30
that the scientific record as it's being
57:32
accumulated is supportive that infection
57:36
with coronavirus likely leads to some
57:38
form of immunity
57:39
dr. pouching yep thank you for the
57:41
question Senator Paul yes you're correct
57:43
that I have said that given what we know
57:45
about the recovery from viruses such as
57:48
corona viruses in general or even any
57:51
infectious disease with very few
57:53
exceptions that when you have anybody
57:55
present it is very likely indicates a
57:58
degree of protection what are your
58:00
thoughts on lockdown as a method of
58:02
dealing with an upper respiratory virus
58:04
my thoughts are just I mean I I'm
58:07
horrified by it it's crazy because we're
58:11
not developing a natural immunity we're
58:14
making people sick that in in that we're
58:18
certainly we protect each other but if
58:21
people are healthy they don't spread
58:23
disease and that's why I mentioned how
58:26
many times in PCR you need to amplify
58:29
that RNA so a mask won't protect a 13
58:35
animal or piece of viral particle from
58:39
spreading through the mask but the mask
58:42
will amplify the viral reservoirs in the
58:46
per
58:46
and it's in so this is so the and the so
58:50
the lockdown is crazy because we don't
58:52
get a natural herd immunity but more
58:55
importantly the mitigation that should
58:58
have happened is interferon alpha and
59:01
hydroxychloroquine and and the serology
59:03
testing all of which costs nothing and
59:06
the healthy people can stay working
59:09
healthy people don't spread disease but
59:12
we are constantly told that this this is
59:15
we are asymptomatic carriers possibly
59:17
well that's not true
59:20
asymptomatic carriers doesn't mean
59:22
you're expressing virus and in the story
59:25
I always use with that shirt you're an
59:27
asymptomatic carrier we have there
59:29
millions of Americans right now who are
59:31
asymptomatic carriers of HIV and XM RVs
59:36
and many other viruses influenza viruses
59:39
because we've been infected before so
59:42
we're asymptomatic carriers but we're
59:44
not spreading disease if we're not sick
59:46
and and I say that and I can say that
59:49
with great confidence because back in
59:51
the 80s the only time I ever isolated
59:56
HIV from saliva were people on their
60:00
deathbed and when Frank Crosetti and
60:03
Bernie poised isolated adult t-cell
60:06
leukemia virus htlv-1 it was from the
60:10
sickest of people with adult t-cell
60:13
leukemia so when you're all the way at
60:16
the end of disease when you're when
60:18
you're so sick those are the people
60:21
expressing the virus so the the sick
60:25
people and the hospitals and it's called
60:27
nosocomial spread you know they should
60:30
be protected not with a mass but given
60:32
oxygen and the nursing staff should be
60:36
wearing the masks that so that the the
60:39
hospital staff isn't exposed to large
60:42
amounts of virus healthy people don't
60:44
spread disease so everything about the
60:47
lockdowns prevents natural herd immunity
60:50
from occurring and we it should have
60:53
never happened it should have been
60:55
everything should have been open
60:56
yesterday the day before last week
60:58
because the evidence
61:00
in this country by those serology tested
61:02
and by anecdotal data is many people at
61:07
least that I talked to every day say I
61:10
had that last November I had that last
61:13
summer oh yeah that was the worst cough
61:15
I ever get but but when you know when I
61:18
recovered you know they're they're
61:20
likely to obey antibodies and some of
61:23
those people we've looked at they're
61:25
actually neutralizing antibodies means
61:27
they can prevent infection in the most
61:30
susceptible so all of these measures
61:33
cost nothing nothing and and none of
61:36
this shut down need ever occur because
61:39
we could have protected people in the
61:40
beginning with a simple very low dose
61:43
type 1 interferon spray and and measures
61:46
of a hydroxychloroquine which has been
61:49
very safe w-h-o essential medicine for
61:52
70 years
61:53
can you just very briefly talk about
61:55
what what you've mentioned before co2
61:58
can you just quickly talk about what are
62:01
the dangers of wearing a mask if you are
62:03
asymptomatic if you're healthy yeah so
62:08
if you're if you're wearing a mask and
62:10
and you're healthy you're breathing in
62:12
your own you know bad air your own toxic
62:16
air so back and forth through the day
62:18
it's very stressful to breathe in toxic
62:21
air you're not getting enough of fresh
62:23
air and oxygen and and just the fact of
62:27
wearing the mask is stressful because
62:29
it's hard to do if you've never been a
62:31
professional doing that it you itch your
62:34
face you you know it's wet its moisture
62:38
it's actually amplifying it it's um it's
62:41
immune suppressive so you're suppressing
62:43
your damping down your own immune system
62:45
your type 1 interferons and you're
62:48
activating your dormant viruses so other
62:50
viruses from within yourself you're
62:53
activating such that now you're
62:56
amplifying them because you're wearing a
62:58
mask you're not protecting anyone and
63:00
and in fact everybody's totally stressed
63:03
out for for all of the reasons they
63:06
should be that you know the people
63:08
aren't intended
63:09
you know it's an upper respiratory
63:10
infection we've known for it
63:14
least a century just washing your hands
63:16
for the medical professionals and and
63:18
protecting yourself from spreading it to
63:21
healthy individuals healthy individuals
63:24
rarely spread these things on the
63:26
subject of mass you made the claim that
63:28
wearing them outside I suppose of acute
63:30
clinical situations is unhelpful that it
63:33
literally activates your own virus
63:35
you're you're getting sick you said from
63:38
your own reactivated coronavirus
63:40
expressions science the journal claims
63:44
to not know what you mean by that can
63:46
you explain in more detail what you're
63:48
suggesting yeah yeah so eight percent of
63:52
our of our genome is a viral and that is
63:57
all of the exposures that we've had and
64:00
we clear and we develop immune responses
64:03
to many are latent like herpes viruses
64:06
like those retroviruses they're dormant
64:09
there your immune system has imbalanced
64:12
they're not expressed if you put on a
64:15
mask and you're exercising and you're
64:19
not breathing air you're actually
64:21
breathing co2 you can become it's called
64:24
lactic acidosis so you become acid and
64:27
you're actually denying yourself oxygen
64:31
hypoxia and then some it's immune
64:34
suppressive it suppresses your very cd4
64:37
positive T cells that adaptive memory
64:40
immunity so you activate the dormant
64:42
because your memory was keeping those
64:44
silent and and this is just this isn't
64:47
new this isn't just me saying it this is
64:50
this is basic immunology and they know
64:53
it activate latent viruses they cause
64:56
disease and the mask is immune
64:59
suppressive and and that that's that's
65:02
clear I don't deny that Tsarskoe v2
65:07
exists and is potentially a pathogenic
65:12
more pathogenic virus to the susceptible
65:15
but the people that were liberally
65:18
called Cove in nineteen without the
65:21
accurate test if you haven't made an
65:24
antibody if you haven't zero converge
65:28
according to your immune system you
65:30
haven't been exposed RNA in your nose is
65:34
not exposure that's why the PCR test is
65:38
a bad one 80% of them were false
65:40
positive I can make that little piece of
65:42
virus and pick out thousands of microbes
65:44
out of your nasal passage that's why you
65:47
have an immune system so people were
65:50
sick because anybody that went in with a
65:52
fever or coughing were called covin 19
65:56
and notice how we don't have influenza
65:59
this year so it's it's known that the
66:03
influenza vaccine over the last few
66:06
years hasn't worked it's not the right
66:09
strain whatever you want to say but the
66:12
government again says go get that
66:14
vaccine anyway and it's the vaccinated
66:17
in two recent papers that are coming out
66:19
that get the flu and you shed that virus
66:22
so the vaccinating or shedding other
66:25
upper respiratory infections and you get
66:29
a respirator respiratory infection was
66:34
called covin 19and and yes this novel
66:37
coronavirus there's no question it's a
66:39
novel coronavirus
66:41
if it got out really the only driving
66:45
force to send it through a hundred and
66:48
ninety countries in a matter of months
66:51
in you know is is through sick people or
66:55
the vaccinated because they're shedding
66:57
other types of viruses to cripple the
67:00
immune system so we do know that in
67:03
Italy in January of 2019 they got that
67:08
four component for influenza virus
67:12
vaccine which had never been used before
67:15
and those vaccines do kill the
67:20
susceptible and they do carry corona
67:22
viruses and you would register as
67:24
positive for the corona virus and
67:27
possibly the novel coronavirus because
67:30
again the vero the the bureau east six
67:34
cell line was transported between Fort
67:37
Detrick and
67:39
on China and North Carolina and other
67:42
places in the world because that's the
67:44
cell line they we grow our our polio
67:48
vaccines our other vaccines and that's
67:50
the one they use to grow these
67:52
coronaviruses so there's not just one
67:54
virus somewhere and we know there are
67:56
many strains and we're seeing those
67:58
mutations in some countries where we're
68:01
different susceptibilities and different
68:03
environmental conditions you know so
68:06
pollution
68:07
cigarette smoking high blood pressure
68:10
diabetes being overweight all of those
68:15
things are risk factors now those are
68:18
the people getting the most severely
68:20
people with other inflammatory diseases
68:23
and that makes sense because
68:25
inflammation the flame is that cytokine
68:29
storm so it's like you took that SARS
68:32
Kove - virus and it was throwing
68:35
gasoline on a fire
68:36
if you throw gasoline on a healthy 20
68:39
year old or on the grass out there
68:41
nothing happens because they don't have
68:44
that fire but Jen that little trigger is
68:47
what we tend to say is the straw that
68:49
broke the camel's back again my
68:52
husband's got COPD and he's a chronic
68:54
obstructive pulmonary disease and he's
68:56
81 years old so so he's the one that is
68:59
at risk every year of an upper
69:02
respiratory infection his his COPD was
69:05
driven by a bacterial infection he got
69:08
as a young man and so but we always
69:11
protect him and the one thing we don't
69:13
do since I met him is we never vaccinate
69:17
him because you don't give somebody
69:19
who's at risk of an upper respiratory
69:21
infection driving a disease another
69:24
upper respiratory infection and that's
69:26
what the vaccine strategy is that's why
69:28
there's nothing about any of the
69:30
candidate back scenes except for the one
69:32
I described earlier and that's why they
69:36
never worked because when you inject the
69:38
blueprint the RNA or DNA of that virus
69:41
or you use I I heard it said oh let's
69:44
just repurpose our polio vaccines I mean
69:47
a horror of Horrors I mean this it
69:49
there's nothing that makes sense in
69:52
anything
69:53
it's happening on a level and it really
69:56
has nothing to do with with the
70:00
administration in any way because Tony
70:03
Fauci stood next to Obama with Ebola of
70:07
14 Zika is 17 you know Bill Curbishley
70:12
anthrax you know Bill Clinton a you know
70:16
swine flu bird flu pig flu what are
70:19
those they're zoonosis they're another
70:21
animal virus that becomes an in a virus
70:25
could be corona virus could be an
70:26
influenza virus are they are they you
70:30
know more quickly evolved man made man
70:33
evolved though no sees those other ones
70:37
are they are they you know
70:39
xenotransplantation as well
70:41
well they well could be but they never
70:43
ended up being much of anything in the
70:45
population because we never we never we
70:50
never took these kind of ridiculous
70:51
draconian measures before so we didn't
70:55
lock down everything in fact that the
70:56
best story is you know for for what
71:00
we're proposing and and many doctors
71:02
around the world good old-fashioned
71:04
medicine and nutrition with you know
71:07
that that our FDA and our CDC are
71:10
stopping us from doing the vitamin C the
71:14
zinc the various nutritional products
71:18
that we that we know work if we did
71:22
those then we would stop the evolution
71:25
of the viruses and and it gets to a can
71:28
of worms we probably don't want to get
71:30
to because the FDA stopped the
71:33
production of interferons for the use of
71:37
preventing zoonosis of coronaviruses
71:41
from animals 40 years ago so we could
71:44
have and what do we do to our animals oh
71:46
we vaccinate them so there's an awful
71:49
lot going on here but the fact of the
71:51
matter is this is 40 years this isn't
71:54
one administration and and this is this
71:58
is 40 years of a practice that's
72:00
literally been controlled by an industry
72:03
that has an interest in money and
72:06
and you know Tony pouchy at the helm
72:09
you know frightening if you will
72:13
presidents and populations that the next
72:16
big pandemic is going to wipe out the
72:19
world like the Spanish flu of 1918